Download PDF by A. Bulikova : Antiphospholipid Syndrome

By A. Bulikova

ISBN-10: 9535105264

ISBN-13: 9789535105268

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The American Society of Hematology. Fig. 2. Electron microscopy analysis of β2GPI. , 2010). Analysis of EM pictures showed that more than 99% of plasma β2GPI was in a closed conformation. These observations suggested that plasma β2GPI circulates in a circular (closed) conformation, whereas after interaction with antibodies β2GPI undergoes a major conformational change into a fishhook-like (open) structure. , 1996). When antibodies toward β2GPI were added to plasma, clotting times prolonged in a β2GPI-dependent way.

Apolipoprotein E receptor 2 is involved in the thrombotic complications in a murine model of the antiphospholipid syndrome. Blood 117:1408-1414. , and Kawane, K. Autoimmunity and the clearance of dead cells. Cell 140:619-630. H. 1987. Fate and behavior of liposomes in vivo: a review of controlling factors. Crit Rev Ther Drug Carrier Syst 3:123-193. , and Kostner, G. 1982. Studies on the possible function of beta 2-glycoprotein-I: influence in the triglyceride metabolism in the rat. Metabolism 31:484-486.

And sepsis patients in the intensive care unit. β2GPI levels returned to normal after recovery, again suggesting an in vivo interaction between β2GPI and LPS. The reduction in β2GPI levels after LPS challenge coincided with an uptake of β2GPI by monocytes. When β2GPI binds to LPS, it changes conformation after which the LPS-β2GPI complex is taken up by monocytes. , 2008). The ability of native β2GPI to inactivate LPS in vivo might offer opportunities to use β2GPI for the treatment of sepsis. β2GPI binds to LPS via domain V of β2GPI.

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Antiphospholipid Syndrome by A. Bulikova

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