Read e-book online Advances in Citrus Nutrition PDF

By Jose M. Garcia-Mina (auth.), Anoop Kumar Srivastava (eds.)

ISBN-10: 9400741707

ISBN-13: 9789400741706

This is a solitary try and streamline the entire attainable details with regards to citrus nutrients, with emphasis on prognosis and administration of nutrient constraints, utilising a number of state-of-art strategies developed globally through the years . whereas doing so care has been taken to incorporate peripheral disciplines in order that the dialogue turns into extra full of life and authoritative. a whole array of unique topics has been properly portrayed with assistance from most up-to-date facts and photographs.

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Plant Biosyst 143:81–96 Gimeno V, Syvertsen JP, Rubio F et al (2010) Growth and mineral nutrition are affected by substrate type and salt stress in seedlings of two contrasting citrus rootstocks. ) grown under salinity. J Plant Physiol 164:728–736 Hafsi C, Romero-Puertas MC, del Río LA (2010) Antioxidative response of Hordeum maritimum L. to potassium deficiency. Acta Physiol Plant 33:193–202 Halliwell B (2006) Reactive species and antioxidants. Redox biology is a fundamental theme of aerobic life.

2005). In citrus, Avsian-Kretchmer et al. (2004) reported that the promoter of the gpx1 gene (encoding for a glutathione peroxidase) responded specifically to hydrogen peroxide. This was evidenced by the fact that ectopic expression of uidA gene under the control of gpx1 promoter was induced by abiotic elicitors (NaCl, sorbitol, mannitol, KCl, and, to a lower extent, by Na2SO4) as well as by H2O2. The addition of catalase or diphenyleneiodonium chloride, an inhibitor of NADPH-oxidase, reduced H2O2-dependent expression of the gpx1 promoter but did not inhibit NaCl induction, suggesting two alternative pathways for hydrogen peroxide signaling: intra- and extracellular (Avsian-Kretchmer et al.

The oxidative reactions that take place in there render large amounts of H2O2 that can cross membranes and affect any surrounding structure or molecule. Excess ROS produced under stress react with many different molecules, as pointed out above, yielding several oxidative by-products such as lipid hydroperoxides (noted as LOO•) that initiate chain reaction cascades with other lipid molecules (unsaturated fatty acids, noted as LOOH) that only end up when a terminator molecule reacts with LOO• (for instance, another LOO•, yielding LOOOOL).

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Advances in Citrus Nutrition by Jose M. Garcia-Mina (auth.), Anoop Kumar Srivastava (eds.)

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